Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance
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Li-Fraumeni syndrome fibroblasts homozygous for p53 mutations are deficient in global DNA repair but exhibit normal transcription-coupled repair and enhanced UV resistance
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External Links
(P356) |
10.1073/PNAS.92.19.8876
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(P698) |
7568035
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(P819) |
1995PNAS...92.8876F
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(P932) |
41070
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(P5875) |
15631153
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