PINK1 defect causes mitochondrial dysfunction, proteasomal deficit and alpha-synuclein aggregation in cell culture models of Parkinson's disease
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PINK1 defect causes mitochondrial dysfunction, proteasomal deficit and alpha-synuclein aggregation in cell culture models of Parkinson's disease
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PINK1 Defect Causes Mitochondrial Dysfunction, Proteasomal Deficit and α-Synuclein Aggregation in Cell Culture Models of Parkinson's Disease |
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External Links
(P356) |
10.1371/JOURNAL.PONE.0004597
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(P698) |
19242547
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(P819) |
2009PLoSO...4.4597L
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(P932) |
2644779
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(P5875) |
24039840
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